Spontaneous necroptosis and autoinflammation are blocked by an inhibitory phosphorylation on MLKL during neonatal development

doi:10.1038/s41422-021-00583-w

	Spontaneous necroptosis and autoinflammation are blocked by an inhibitory phosphorylation on MLKL during neonatal development
	Zhu, Xinxin1,2,3 ; Yang, Na 2,3; Yang, Yu 2,3; Yuan, Feiyang1,2,3 ; Yu, Dandan1,3 ; Zhang, Yu1,2,3 ; Shu, Zhaoqian1,3 ; Nan, Ning1,2,3 ; Hu, Hong1 ; Liu, Xiaoyan1 ; Chen, She 4,5; Sun, Liming 2,3; Wang, Huayi1
	2022-04
发表期刊	CELL RESEARCH (IF:28.1[JCR-2023],36.4[5-Year])
ISSN	1001-0602
EISSN	1748-7838
DOI	10.1038/s41422-021-00583-w
URL	查看原文
收录类别	SCIE
语种	英语
WOS研究方向	Cell Biology
WOS类目	Cell Biology
WOS记录号	WOS:000713931100001
出版者	SPRINGERNATURE
原始文献类型	Letter; Early Access
引用统计	被引频次：22[WOS] [WOS记录] [WOS相关记录]
文献类型	期刊论文
条目标识符	https://kms.shanghaitech.edu.cn/handle/2MSLDSTB/128582
专题	生命科学与技术学院_博士生生命科学与技术学院_PI研究组_王华翌组生命科学与技术学院_PI研究组_黄行许组生命科学与技术学院_硕士生生命科学与技术学院_本科生
通讯作者	Sun, Liming; Wang, Huayi
作者单位	1.ShanghaiTech Univ, Sch Life Sci & Technol, Shanghai, Peoples R China; 2.Univ Chinese Acad Sci, Shanghai Inst Biochem & Cell Biol, CAS Ctr Excellence Mol Cell Sci, State Key Lab Cell Biol,Chinese Acad Sci, Shanghai, Peoples R China; 3.Univ Chinese Acad Sci, Beijing, Peoples R China; 4.Natl Inst Biol Sci, Beijing, Peoples R China; 5.Tsinghua Univ, Tsinghua Inst Multidisciplinary Biomed Res, Beijing, Peoples R China
第一作者单位	生命科学与技术学院
通讯作者单位	生命科学与技术学院
第一作者的第一单位	生命科学与技术学院
推荐引用方式 GB/T 7714	Zhu, Xinxin,Yang, Na,Yang, Yu,et al. Spontaneous necroptosis and autoinflammation are blocked by an inhibitory phosphorylation on MLKL during neonatal development[J]. CELL RESEARCH,2022.
APA	Zhu, Xinxin.,Yang, Na.,Yang, Yu.,Yuan, Feiyang.,Yu, Dandan.,...&Wang, Huayi.(2022).Spontaneous necroptosis and autoinflammation are blocked by an inhibitory phosphorylation on MLKL during neonatal development.CELL RESEARCH.
MLA	Zhu, Xinxin,et al."Spontaneous necroptosis and autoinflammation are blocked by an inhibitory phosphorylation on MLKL during neonatal development".CELL RESEARCH (2022).